Association Between MC-2 Peptide and Hepatic Perfusion and Liver Injury Following Resuscitated Hemorrhagic Shock | Zendy

Paul J. Matheson | Zendy; Rafael Fernandez-Botrán | Zendy; Jason W. Smith | Zendy; Samuel A. Matheson | Zendy; Cynthia D. Downard | Zendy; Craig J. McClain | Zendy; R. Neal Garrison | Zendy

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Association Between MC-2 Peptide and Hepatic Perfusion and Liver Injury Following Resuscitated Hemorrhagic Shock

Author(s) -

Paul J. Matheson,

Rafael Fernandez-Botrán,

Jason W. Smith,

Samuel A. Matheson,

Cynthia D. Downard,

Craig J. McClain,

R. Neal Garrison

Publication year - 2015

Publication title -

jama surgery

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.757

H-Index - 176

eISSN - 2168-6262

pISSN - 2168-6254

DOI - 10.1001/jamasurg.2015.4050

Subject(s) - medicine , resuscitation , shock (circulatory) , proinflammatory cytokine , liver injury , organ dysfunction , perfusion , hemodynamics , endocrinology , gastroenterology , anesthesia , inflammation , sepsis

Hemorrhagic shock (HS) due to trauma remains a major cause of morbidity and mortality in the United States, despite continuing progression of advanced life support and treatment. Trauma is the third most common cause of death worldwide and is the leading cause of death in the 1- to 44-year-old age group. Hemorrhagic shock often progresses to multiple organ failure despite conventional resuscitation (CR) that restores central hemodynamics.

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