Neurochemical Aftermath of Repetitive Mild Traumatic Brain Injury | Zendy

Pashtun Shahim | Zendy; Yelverton Tegner | Zendy; Bengt Gustafsson | Zendy; Magnus Gren | Zendy; Johan Ärlig | Zendy; Martin Olsson | Zendy; Niklas Lehto | Zendy; Åsa Engström | Zendy; Kina Höglund | Zendy; Erik Portelius | Zendy; Henrik Zetterberg | Zendy; Kaj Blennow | Zendy

AI Assistant Blog Pricing

Home ZAIA Blog

Open Access

Neurochemical Aftermath of Repetitive Mild Traumatic Brain Injury

Author(s) -

Pashtun Shahim,

Yelverton Tegner,

Bengt Gustafsson,

Magnus Gren,

Johan Ärlig,

Martin Olsson,

Niklas Lehto,

Åsa Engström,

Kina Höglund,

Erik Portelius,

Henrik Zetterberg,

Kaj Blennow

Publication year - 2016

Publication title -

jama neurology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 5.298

H-Index - 231

eISSN - 2168-6157

pISSN - 2168-6149

DOI - 10.1001/jamaneurol.2016.2038

Subject(s) - traumatic brain injury , rivermead post concussion symptoms questionnaire , chronic traumatic encephalopathy , concussion , medicine , cerebrospinal fluid , glial fibrillary acidic protein , poison control , psychology , pediatrics , injury prevention , psychiatry , immunohistochemistry , environmental health

Evidence is accumulating that repeated mild traumatic brain injury (mTBI) incidents can lead to persistent, long-term debilitating symptoms and in some cases a progressive neurodegenerative condition referred to as chronic traumatic encephalopathy. However, to our knowledge, there are no objective tools to examine to which degree persistent symptoms after mTBI are caused by neuronal injury.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research