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A Case of Sweet Syndrome Associated With Human Granulocytic Anaplasmosis
Author(s) -
Charles L. Halasz,
George Niedt,
Caroline P. Kurtz,
Diana G. Scorpio,
Johan Bakken,
J. Stephen Dumler
Publication year - 2005
Publication title -
archives of dermatology
Language(s) - English
Resource type - Journals
eISSN - 1538-3652
pISSN - 0003-987X
DOI - 10.1001/archderm.141.7.887
Subject(s) - anaplasma phagocytophilum , medicine , leukopenia , immunology , serology , polymerase chain reaction , pathology , dermatology , gene , antibody , biology , biochemistry , chemotherapy , borrelia burgdorferi
Acute febrile neutrophilic dermatosis, or Sweet syndrome (SS), is a condition that is presumed to be triggered by infectious disease agents. We report a case of SS associated with human granulocytic anaplasmosis (HGA), which is of interest because Anaplasma phagocytophilum infects, multiplies in, and disrupts the function of neutrophils, the key infiltrating cell in SS.

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