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rable to levels in healthy controls (2,727.4 ± 430.9 pg/ ml). When classified by type of skin lesions, serum sHVEM levels in patients with CTCL with early skin lesions (patch and plaque), tumours, and erythroderma were 2,708.0 ± 609.7 pg/ml, 3,307.3 ± 580.1 pg/ml, and 3,323.2 ± 535.2 pg/ml, respectively. The levels in patients with tumours and those with erythroderma were signifi cantly higher than those in healthy controls (Fig. 1a). Serum LIGHT levels in patients with CTCL (134.6 ± 165.0 pg/ml) were significantly higher than those in healthy controls (61.2 ± 28.7 pg/ml; p < 0.05). In patients with early skin lesions, tumours, and erythro derma, serum LIGHT levels were 87.2 ± 96.1 pg/ml, 146.5 ± 121.9 pg/ ml, and 313.8 ± 340.3 pg/ml, respectively. The levels in patients with tumours and those with erythroderma were significantly higher than those in healthy controls (Fig. 1b). Serum sHVEM levels correlated significantly with serum LIGHT levels (Fig. 1c), serum sIL-2R levels (Fig. 1d), the percentages and numbers of eosinophils (Fig. 1e, f), and serum IL-4 and IL-10 levels (Fig. 1g, h). Serum sHVEM levels did not show significant correlations with age, sex, or serum LDH and IgE levels. Serum LIGHT levels did not correlate significantly with laboratory data described above except serum sHVEM levels.

Serum-soluble Herpes Virus Entry Mediator Levels Reflect Disease Severity and Th2 Environment in Cutaneous T Cell Lymphoma