Open Access<p>HBXIP Regulates Gastric Cancer Glucose Metabolism and Malignancy Through PI3K/AKT and p53 Signaling</p>
Author(s) -
Lei Qiu,
Feng Lu,
Lili Zhang,
Gang Wang,
Rui Geng,
Yi Miao
Publication year - 2020
Publication title -
oncotargets and therapy
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.054
H-Index - 60
ISSN - 1178-6930
DOI - 10.2147/ott.s243250
Subject(s) - cell growth , apoptosis , cancer research , in vivo , pi3k/akt/mtor pathway , protein kinase b , carcinogenesis , biology , cell cycle , cell migration , cell , tunel assay , cancer , chemistry , microbiology and biotechnology , biochemistry , genetics
Hepatitis B X-interacting protein (HBXIP) overexpression is related to the progression of multiple cancers. However, its role in gastric cancer (GC) remains unclear.