Open AccessDo People Live at Sea Level and the Dead Sea Level Have Different Patterns of Anti-Hypertensive Drugs
Author(s) -
Arwa Ali Alhroot,
Alen Fareed Alfanek,
Muna Alkhushman,
Amal Saad Eldeen Aljariri,
Diana A. Ibrahim,
Farah Mahmoud Abu Abeeleh,
Jaafar Abu Abeeleh,
Ahed J Alkhatib,
Mahmoud Abu Abeeleh
Publication year - 2017
Publication title -
european scientific journal
Language(s) - English
Resource type - Journals
eISSN - 1857-7881
pISSN - 1857-7431
DOI - 10.19044/esj.2017.v13n3p416
Subject(s) - blood pressure , dead sea , medicine , sea level , demography , population , population study , cross sectional study , geography , environmental health , cartography , oceanography , pathology , sociology , geology
Background: people live at various areas of sea level may have different patterns of anti-hypertensive drugs. Such a relation has never been reported in Jordan. Study objectives: the current study investigated how the sea level will impact the prevalence of hypertension in these areas, and how will affect the pharmacological properties of such a population. Methodology: a cross-sectional study design was involved to collect data from study participants. A total of 1000 participants were randomly selected from the two study areas. 500 participants from each. Participants were matched for age and gender. Blood pressure were measured for all participants. Blood samples were withdrawn to investigate the level of angiotensin II. Data was collected through organizing a working excel sheet and was further analyzed through using SPSS version 20. Data was presented as means, standard deviations, frequencies and percentages. The relationships between variables were examined using independent T-test. Significance was measured at an alpha 0.05). Conclusions: although the level of angiotensin II was not significantly varied between the study areas, but its trend was to be higher in the Sea level area. It was surprised to have higher levels of SBP and MBP in the Dead Sea rather than the Seal level area. It can be implied that the therapeutic options of hypertensive drugs follow different patterns independent of angiotensin II pathways.