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Activating BRAF mutations, most commonly BRAF V600E , are a major oncogenic driver of many cancers. We explored whether BRAF V600E promotes radiation resistance and whether selectively targeting BRAF V600E with a BRAF inhibitor (vemurafenib, BRAFi) sensitizes BRAF V600E thyroid cancer cells to radiotherapy.

clinical cancer research

Inhibiting BRAF Oncogene–Mediated Radioresistance Effectively Radiosensitizes BRAFV600E-Mutant Thyroid Cancer Cells by Constraining DNA Double-Strand Break Repair