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Three types of mutants (mel − ) unable to metabolize the α-d -galactoside, melibiose, were derived fromEscherichia coli K-12. One type lacked α-galactosidase; another lacked a specific transport system, termed thiomethylgalactoside (TMG) permease II; and the third lacked both of these functions. The mutational sites were genetically mapped by recombination frequency with different markers and by determination of chromosomal transfer in interrupted-mating experiments. All threemel − mutant types mapped in a cluster near to themetA marker on theE. coli chromosome and were cotransducible. Induction studies revealed that the three α-d -galactosides, melibiose, melibiitol, and galactinol, induced α-galactosidase and TMG permease II coordinately;d -galactose also induced them but only in a galactokinaseless mutant. These data suggest that α-galactosidase and TMG permease II may be components of a common operon.

Analysis of Melibiose Mutants Deficient in α-Galactosidase and Thiomethylgalactoside Permease II in Escherichia coli K-12