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Branched- and straight-chain alkanes are metabolized byBrevibacterium erythrogenes by means of two distinct pathways. Normal alkanes (e.g.,n -pentadecane) are degraded, after terminal oxidation, by the beta-oxidation system operational in fatty acid catabolism. Branched alkanes like pristane (2,6,10,14-tetramethylpentadecane) and 2-methylundecane are degraded as dicarboxylic acids, which also undergo beta-oxidation. Pristane-derived intermediates are observed to accumulate, with time, as a series of dicarboxylic acids. This dicarboxylic acid pathway is not observed in the presence of normal alkanes. Release of14 CO2 from [1-14 C]pristane is delayed, or entirely inhibited, in the presence ofn -hexadecane, whereas CO2 release fromn -hexadecane remains unaffected. These results suggest an inducible dicarboxylic acid pathway for degradation of branched-chain alkanes.

Hydrocarbon Metabolism by Brevibacterium erythrogenes : Normal and Branched Alkanes