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Intracellular and cell-to-cell spread of Listeria monocytogenes has been considered exclusively actin dependent. By immunocytochemical techniques, we provide evidence for an involvement of inhibitory G proteins and tubulin in "comet tail" formation in L. monocytogenes-infected mouse hepatocytes.

Involvement of tubulin and inhibitory G proteins in the interaction of Listeria monocytogenes with mouse hepatocytes