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Ralstonia solanacearum  releases a set of effectors into plant cells that modify the host defence reaction. The role of the effector protein RipI during infection has not been elucidated. In this study, we demonstrated that transient overexpression of RipI induces the hypersensitive response (HR), up‐regulating the HR marker gene  hin1 , in  Nicotiana benthamiana . Deletion of  R. solanacearum ripI  led to increased virulence in tomato ( Solanum lycopersicum ) plants. Through yeast two‐hybrid and pull‐down assays, we identified an interaction between the  N. benthamiana  transcription factor bHLH93 and RipI, both of which could be localized in the nucleus of  Arabidopsis  protoplasts .  Silencing of  bHLH93  markedly attenuated the RipI‐induced HR and induced expression of the  PDF1.2  defence gene. These data demonstrate that the  R. solanacearum  effector RipI induces a host defence reaction by interacting with the bHLH93 transcription factor.

The Ralstonia solanacearum effector RipI induces a defence reaction by interacting with the bHLH93 transcription factor in Nicotiana benthamiana