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Glucose‐ or insulin resistance‐mediated β‐cell replication serves as a useful physiological model of β‐cell mass expansion to study the pathophysiology of type 2 diabetes. Lakshmipathi et al. (2016) report that PKCζ activation is crucial for compensatory β‐cell proliferation induced by glucose stimulation or high‐fat diet‐feeding.

Glucose‐ or insulin resistance‐mediated β‐cell replication: PKC ζ integrates the proliferative signaling