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ONO ‐2506 inhibits spike–wave discharges in a genetic animal model without affecting traditional convulsive tests via gliotransmission regulation
Author(s) -
Yamamura Satoshi,
Hoshikawa Masamitsu,
Dai Kato,
Saito Hiromitsu,
Suzuki Noboru,
Niwa Osamu,
Okada Motohiro
Publication year - 2013
Publication title -
british journal of pharmacology
Language(s) - Uncategorized
Resource type - Journals
SCImago Journal Rank - 2.432
H-Index - 211
eISSN - 1476-5381
pISSN - 0007-1188
DOI - 10.1111/j.1476-5381.2012.02132.x
Subject(s) - kynurenic acid , biology , neurotransmission , neuroscience , glutamate receptor , excitatory postsynaptic potential , inhibitory postsynaptic potential , depolarization , pentylenetetrazol , tiagabine , gaba receptor antagonist , ampa receptor , epilepsy , gamma aminobutyric acid , pharmacology , bicuculline , biophysics , gabaa receptor , biochemistry , anticonvulsant , receptor
This article is commented on by Onat, pp. 1086‐1087 of this issue .
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