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When the Wnt pathway is off duty, the transcription factor FOXP2 can up‐ and downregulate the transcription of a set of genes including Wnt‐related targets. Upon activation of the Wnt pathway, β‐catenin via direct interaction with FOXP2 can modulate its transcriptional activity in a TCF/LEF‐dependent and/or TCF/LEF‐independent manner.

β‐catenin regulates FOXP2 transcriptional activity via multiple binding sites