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the immune system in psoriatic patients, whereby T helper 17 cells or innate lymphoid cell 3 systems become overactive to compensate, even though the defect cannot be detected clinically. The Koebner phenomenon may explain some of the distribution of the plaques as the increased expression of an antimicrobial peptide, cathelicidin, in psoriatic lesional epidermis is one of the provoking causes of inflammatory cascades.

Agminated Spitz naevi or metastatic spitzoid melanoma?