English (United Kingdom)

https://curated-unify.zendy.io/wp-json/zendy-region/v1/featured_content/oa?rat=en

https://curated-unify.zendy.io/wp-json/zendy-region/v1/highlighted_journal/

Global: Zendy Plus annual

Presents the access of premium content as premium feature

Premium Content

Presents the keyphrase highlighting as premium feature

Keyphrase Highlighting

Presents the summarisation as premium feature

Summarisation

Insights

Presents the pdf analysis as premium feature

PDF Analysis

Presents the zaia usage as premium feature

ZAIA

Global: Zendy Plus monthly

Global: Zendy Tools annual

Global: Zendy Tools monthly

Global: Zendy Free Plan

Spatial learning across many species is impaired by lesions in thehippocampus, a subcortical brain structure whose cellular composition changessubstantially over its 5 to 6 week lifetime from mainly excitatory neuronsduring development to equal proportions of inhibitory interneurons (gammaAmp/Arcs) as well as pyramidal cells early in life, but which later oncomprises only about 10% Arches+ projection spiny cortical projecting principalcells that are located within discrete cytoarchitectonic patches known as CA3or just hilus granular layer 2 (CG2). While other structures may contributeimportantly in certain situations e.g., perirhinal cortex when using visualcues with no reference frame for location), these remaining cell types alsochange their proportion through time with gamma APs forming 30 to 35 percent),fast firing parvalbumin immunoreactive basket or axoaxonic synapses intosomatic spines at 40 days after birth (DABs formed into spine membranes by 15DAB), and CG2 pyramids forming relatively slowly but strengthening synapsesonto nearby dendritic shafts made up largely (>80 %) by GABAergic terminals(+spines) until then that were still synaptically silent.

Structural Correlates Of Spatial Navigation And Memory Formation