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In the context of increased number of works published on multisensory andcross-modal effects, we review a cortical multisensory syndrome (called centralsyndrome) associated with a unilateral parieto-occipital lesion in a ratherunspecific (or multisensory) zone of the cortex.  The patients with this syndrome suffered from bilateral and symmetricmultisensory disorders dependent on the extent of nervous mass lost and theintensity of the stimulus. They also presented cross-modal effects. A key pointis the similitude of this syndrome with a normal state, since this syndromewould be the result of a scale reduction in brain excitability. The firstqualities lost when the nervous excitation diminishes are the most complexones, following allometric laws proper of a dynamic system.  The inverted perception (visual, tactile, auditive) in this syndrome iscompared to other cases of visual inversion reported in the literature. Wefocus on the capability of improving perception by intensifying the stimulus orby means of another type of stimulus (cross-modal), muscular effort being oneof the most efficient and least known means. This capability is greater whennervous excitability deficit (lesion) is greater and when the primary stimulusis weaker. Thus, in a normal subject, this capability is much weaker althoughperceptible for functions with high excitability demand. We also review theproposed scheme of functional cortical gradients whereby the specificity of thecortex is distributed with a continuous variation leading to a brain dynamicsmodel accounting for multisensory or cross-modal interactions. Perception data(including cross-modal effects) in this syndrome are fitted using Stevens'power law which we relate to the allometric scaling power laws dependent on theactive neural mass, which seem to be the laws governing many biological neuralnetworks.

Nervous Excitability dynamics in a multisensory syndrome and its similitude with a normal state. Scaling Laws