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The model for cAMP-dependent synaptic plasticity relates the characterizationof a noradrenaline-stimulated adenylyl cyclase with DNA unzipping. Specificproteins condition cAMP insertion in specific sites of the DNA structure todirect cellular and synaptic differentiation in brain tissues, also providingcoding. Metabolic-dependent ATP binding of Mg2+, could control feedback byinactivating AC dependent formation of cAMP. The level of cAMP and cGMP, whichcould be assayed in red cells and cerebrospinal fluid, allows a clinical labdiagnostic improvement. Also, provides a relationship of best fitting to cAMPcontrol by binding to DNA. The cAMP level allows the prediction of an inverserelationship between neurodegeneration and cancer. The latter, could becharacterized by uncontrolled proliferation, whereas metabolic dominance bystress over a long period of time, may deplete cerebral cAMP.

The integrated model of cAMP-dependent DNA expression reveals an inverse relationship between cancer and neurodegeneration