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Memristors have been suggested as neuromorphic computing elements. Spike-timedependent plasticity and the Hodgkin-Huxley model of the neuron have both beenmodelled effectively by memristor theory. The d.c. response of the memristor isa current spike. Based on these three facts we suggest that memristors arewell-placed to interface directly with neurons. In this paper we show thatconnecting a spiking memristor network to spiking neuronal cells causes achange in the memristor network dynamics by: removing the memristor spikes,which we show is due to the effects of connection to aqueous medium; causing achange in current decay rate consistent with a change in memristor state;presenting more-linear $I-t$ dynamics; and increasing the memristor spikingrate, as a consequence of interaction with the spiking neurons. Thisdemonstrates that neurons are capable of communicating directly withmemristors, without the need for computer translation.

Connecting Spiking Neurons to a Spiking Memristor Network Changes the Memristor Dynamics