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Unstable angina is common clinical manifestation of atherosclerosis. However,the detailed pathogenesis of unstable angina is still not known. Here, Ipropose that unstable angina is a mixed TH17 and TH1 immune disorder. By usingmicroarray analysis, I find out that TH1 and TH17 related cytokine, cytokinereceptor, chemokines, complement, immune-related transcription factors,anti-bacterial genes, Toll-like receptors, and heat shock proteins are allup-regulated in peripheral leukocytes of unstable angina. In addition,H-ATPase, glycolytic genes, platelet and RBC related genes are alsoup-regulated in peripheral leukocytes of during unstable angina. It alsoimplies that atherosclerosis is a mixed TH17 and TH1 autoimmune disease. If weknow the etiology of unstable angina as well as atherosclerosis better, we canhave better methods to control and prevent this detrimental illness.

Unstable Angina is a syndrome correlated to mixed Th17 and Th1 immune disorder