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A review of recent investigations into pathogenesis of psoriasis summarizingdata on the relationship between the presence beta-streptococci in the organismand the cutaneous immune reaction. Results of the studies on gastrointestinalpathology in psoriatic patients are presented. The authors propose a hypothesisthat advocates the primary origin of psoriatic gastrointestinal pathology andsecondary nature of skin manifestations. A chronic plaque psoriasis model isdeveloped on the basis of the assumption on the key role of two psorafactors,i.e. hyperpermeability of intestinal walls for certain proteins andincorporation of beta-streptococci in the microbiocenosis of intestinal mucosa.The validity of the model is confirmed by the results of practical clinicalwork.

Psoriasis as a consequence of incorporation of beta-streptococci into  the microbiocenosis of highly permeable intestines (a pathogenic concept).  Russian edition