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Aims: The objective of present work is to investigate metabolic alterations associated with heart failure, particularly one of its manifestations, a sustained hypocalcemiathatcauses hemodynamic changes contributed to subsequent myocardial injury. Comparative study was carried outusing experimental modelsof pancreatic necrosis (PN) and crush syndrome (CS) accompanied by cardiac damage down tomyocardial infarction. Study design: Wistaradult male rats randomly divided into groups (n=12/group). The controls are healthy intact animals. The pancreatic necrosis (PN) and crush syndrome (CS) groups were then randomly subdivided: PN group -into 3, 24 and 72 h groups

Alterations in Calcium-Binding Properties of Sarcoplasmic Reticulum Membrane Proteins Following Cardiac Injury