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Cutaneous vasculitides include an extensive and diverse cluster of diseases affecting the tegumentary blood vessels; these are clinically manifested as polymorphic skin lesions, including palpable purpura, as well as urticarial and necrotic-ulcerative lesions. Sometimes extracutaneous involvement occurs [1]. Leukocytoclastic vasculitis (LV) was formerly known as hypersensitivity vasculitis. LV can have an acute form and a chronic presentation [2]. Cutaneous leukocytoclastic angiitis and urticarial vasculitis, which are the two most common variations of this disease cluster, are frequently idiopathic but may also be triggered by diverse agents, including drugs and infections, or may be as part of systemic disorders, predominantly systemic lupus erythematosus (SLE) [1]. Vasculitic skin lesions can also occur during the chronicrelapsing course of systemic vasculitides, including Wegener’s granulomatosis (WG). These vasculitides often share clinical and immunohistopathologic features [1,2]. The vasculitides are associated with blood vessel inflammation that can cause internal organ damage [1-3].

An extraordinary case of oral leukocytoclastic vasculitis demonstrating igd deposition around dermal vessels and within mucosal keratinocytes