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Onychomadesis is characterized by spontaneous detachment of the nail plate from the nail matrix, in which detachment begins at the proximal end of the nail bed, although not always, consequently leading to nail shedding. A vast variety of health conditions, mainly including infections and autoimmune diseases have been implicated in the etiology of onychomadesis [1,2]. It is well-known that thyroid diseases have significant cutaneous manifestations, some of which are distinctly linked with nails [3]. Thyroid acropachy is the utmost cutaneous manifestation of autoimmune thyroid disease, which is characterized by clubbing of fingers and toenails, swollen digits along with subperiosteal new bone formation [4-10]. Here, we describe a patient with onychomadesis associated with autoimmune thyroid disease, whom clinical features mimic those of thyroid acropachy. To our knowledge, this is the first case to be published in which the clinical findings favored the diagnosis of onychomadesis associated with autoimmune thyroid disease. CASE REPORT

Onychomadesis secondary to autoimmune thyroid disease mimicking thyroid acropachy