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Anaplastic thyroid cancer (ATC) is highly aggressive and has a poor therapeutic response and leads to high mortality. It has been shown that activation of intracellular c-Jun N-ter‐ minal kinase (JNK) and c-ABL signaling pathways is one of the manifestations of the highly resistant response to radiotherapy in ATC. Pharmacological inhibition of these pathways in combination with radiotherapy is a potential treatment modality of ATC.

Pharmacological Inhibition of Intracellular Signaling Pathways in Radioresistant Anaplastic Thyroid Cancer