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Diabetic ketoacidosis (DKA) is considered a predominantly acute type 1 diabetic complica‐ tion, although it may occur in type 2 diabetes as well, particularly in patients who already have a decreased insulin secretion capacity. Stress –induced burst in catecholamine and ACTH secretion in acute myocardial infarction (AMI) promotes release of free fatty acids and their hepatic and muscular tissue utilization. The impairment in insulin-mediated intra‐ cellular glucose influx owing to the absent or insufficient pancreatic insulin secretion is the prerequisite for the occurrence of diabetic ketoacidosis.

Distinctive Characteristics and Specific Management of Diabetic Ketoacidosis in Patients with Acute Myocardial Infarction, Stroke and Renal Failure