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The lung is the human organ most susceptible to oxidative damage. The transition from fetal, a low-oxygen environment, to FiO2: 0.21, induces a relative degree of oxidative stress for all newborns, but especially in the case of preterm neonates whose lung development has been interrupted. A supplementary supply of oxygen (hyperoxia) is often used to treat preterm newborns and these infants may not yet be prepared to protect their lungs from oxidative injury.

Effects of Oxidative Stress and Antenatal Corticosteroids on the Pulmonary Expression of Vascular Endothelial Growth Factor (VEGF) and Alveolarization