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The excessive proliferation and migration of vascular smooth muscle cells (SMCs) and angiogenesis of endothelial cells (ECs) participate in the growth and instability of atherosclerotic plaques. It is unclear whether Jun N-terminal kinase (JNK) is pro-or anti-atherogenic.

Jun N-Terminal Kinase Inhibitor Blocks Angiogenesis by Blocking VEGF Secretion and an MMP Pathway