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N-acetylcysteine (NAC) has antioxidant and anti-inflammatory properties. To explore the mechanisms underlying atherosclerotic plaque stabilization induced by NAC, we examined the effects of NAC administration in apoE-deficient mice on the expression of the receptor of advanced glycation end products (RAGE), matrix metalloproteinases (MMPs) and the activation of nuclear factor kappa B (NF-κB) in atherosclerotic plaques.

journal of atherosclerosis and thrombosis

The Antioxidant N-Acetylcysteine Promotes Atherosclerotic Plaque Stabilization Through Suppression of RAGE, MMPs and NF-&amp;kappa;B in ApoE-Deficient Mice