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runs varyingly in different laboratories (3). As stated by the authors, HPLC coupled to mass spectrometric detection (LC-MS/MS) is not widely available, and the equipment is comparatively expensive (4). So, HPLC was the preferable method for us to detect ADMA. Intravascular ultrasonography is a validated and superior method when compared with coronary angiography to determine neointimal tissue burden, assessment of lesion significance, and stent restenosis (5). But, in our country conditions, it is not feasible to evaluate stent restenosis for every patient because of its cost and low availability. There are so many relevant biomarkers known for stent restenosis (6), but it is not feasible to evaluate all of them in one study protocol. Our aim was to evaluate if ADMA predicts stent restenosis beyond classic predictors or not. In our study, we concluded that plasma ADMA level may be used as a novel marker for stent restenosis beyond the classic stent restenosis markers. However, as we stated in our study, this result should be confirmed by larger, prospective, and controlled studies.

Should systemic thrombolytic therapy be considered a first-line treatment in acute pulmonary embolism?