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We studied the interactions between electromagnetic fields (EMF), the Hydrogen isotope (H1) and the biophysical process that precedes the biochemical expression of the cell’s energetic mechanisms. The hydrogen isotope, being the simplest atom, its manipulation is more technically feasible. In addition of its importance in metabolic energy driving reactions due to its reductive power, the hydrogen atom seems to be a necessary cofactor and driving force to induce, foster and/or restore oxidative phosphorylation in the cancer cell. Normal cells require a higher level of energy to attain and sustain the order, communication and direction necessary for the maintenance of the differentiated state. When cellular aerobic energy mechanisms are damaged, the energy is not enough to sustain the complexity and specificity of differentiation and therefore it can only either die or transform to a simpler state (less differentiated) that requires less energy in order to survive. This is the genesis of the malignant state. The interaction of the H1 with the EMF produces a change in the permitivity of the mitochondrial membrane that favors the release of energy allowing for the oxidative phosphorylation to proceed. We propose that this interaction of the EMF and the H1 can be manipulated in such a way that it would be capable of clearing interferences of fields that may favor transformation to the malignant state.

RETRACTED: Biophysics and Cancer: The Electromagnetic Fields Produced by the Mitochondria and Its Effect on the Cell’s Metabolic Regulation