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We, and several other research groups, quickly realized that the best mimicking clinical situation characterized by a delayed onset of intracranial arterial vasospasm has been a non-human primate model introduced by Espinoza et al. in the Bryce Weir laboratory in 1984 [2]. Over the decades of intensive, multicenter experimental effort using a non-human primate, but also other models, elucidated many mechanisms underlying deleterious effects of aSAH in some people as well as initiated a paradigm shift in goals for adequate treatment development.

Commentary on a Non-Human Primate Model of Aneurismal Subarachnoid Hemorrhage