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Although epidermal growth factor receptor (EGFR), v-Ki-ras2 Kirsten rat sarcoma viral oncogene (KRAS), and anaplastic lymphoma kinase (ALK) mutations in non-small cell lung cancer (NSCLC) were thought to be mutually exclusive, some tumors harbor concomitant mutations. Discovering a driver mutation on the basis of morphologic features and therapeutic responses with mutation analysis can be used to understand pathogenesis and predict resistance in targeted therapy.

journal of pathology and translational medicine

Non-small Cell Lung Cancer with Concomitant &lt;i&gt;EGFR&lt;/i&gt;, &lt;i&gt;KRAS&lt;/i&gt;, and &lt;i&gt;ALK&lt;/i&gt; Mutation: Clinicopathologic Features of 12 Cases