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Despite the enduring status of ischemic stroke as a major cause of morbidity and mortality worldwide, a deficiency of effective therapies currently exists to treat this deadly disease. It is therefore essential that research continues to be conducted into the molecular mechanisms that precipitate injury following strokes. These mechanisms may, one further elucidated, yield the therapeutic targets that have eluded clinical practice thus far.

Hypoxia-inducible factor-1 α and RIP3 triggers NLRP3 inflammasome in ischemic stroke