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Alzheimer's disease (AD) is a neurodegenerative disorder which is remarkably characterized by pathological hallmarks that include neurofibrillary tangles, neuronal loss extracellular senile plaques containing aggregated amyloid beta (Aβ), and neurofibrillary tangles composed of the hyper- phosphorylated form of the microtubule protein tau. It is the most common form of dementia which is characterized by severe neurodegenerative changes such as loss of neurons and synapses in brain (Kamat et al., 2014). Several well-known mutations that lead to early-onset familial AD exist. However, these circumstances only reason for a few percentages of total AD cases.

Streptozotocin induced Alzheimer′s disease like changes and the underlying neural degeneration and regeneration mechanism