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Sir, I read with interest the article by Lakshmi et al.[1] In this article the authors have suggested that both type I hypersensitivity and type IV hypersensitivity are responsible for dermatitis in parthenium dermatitis patients, and they have tried to demonstrate this by doing prick test and serum IgE levels in these patients. The majority of their patients were atopics. It is well known that atopic individuals are more susceptible to develop allergic response to various antigenic stimuli and have elevated IgE levels.[2] Immunologic abnormalities of type I and type IV reactions have been described in patients with atopic dermatitis.[3] Immunologic triggers are aeroallergens, food allergens, microbial products, autoallergens and contact allergens. They enhance IgE production by B lymphocytes with an increased secretion of interleukin 4, interleukin 5 and interleukin 13.[2]

Type I hypersensitivity to <i> Parthenium hysterophorus</i> in patients with parthenium dermatitis