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To the editor. Genotyping of CMV has mainly focused on gB, gN, and gH, which play a role in virus entry and may influence the infectivity or pathogenicity of CMV.1,2 It has been hypothesized that genetic variation among CMV strains may underlie strain-specific clinical manifestations. Our previous research revealed that there might be a potential association between the genotypes of CMV and neonatal thrombocytopenia, and the detection of some specific genotypes might be indicative of severe manifestations in infants with CMV infection.3,4 However, the study design and the criteria to define the study population (congenital and noncongenital cases) and the setting of the control group (CMV-associated thrombocytopenia and nonthrombocytopenic cases) were not clearly established. For this reason, we included patients classified on more unambiguous criteria, and the clinical data collected were complete and thoroughly detailed, which allows us to assess the association between genotypes and the outcome in the non-congenital population.

mediterranean journal of hematology and infectious diseases

Cytomegalovirus genotype distribution among postnatally infected infants: association of glycoprotein B, glycoprotein N and glycoprotein H types with CMV-associated thrombocytopenia