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Cortisol has been implicated in hypertension and latelyreported to be regulated at the pre-receptor level by the 11sHSD1 enzyme, which converts cortisone (E)to cortisol (F). Over expression of this enzyme in adipose tissue could determine an increase in availablecortisol that interacts with the mineralocorticoid receptor (MR) in renal, brain and heart tissue, leadingto similar hypertensive effects as in 11sHSD2 impaired patients. Several polymorphisms have beenreported in HSD11B1 gene (CA15, CA19 and InsA83557), which could modify HSD11B1 geneexpression or activity.

Regiones polimórficas del gen 11ß-hidroxiesteroide deshidrogenasa tipo 1 (11ßHSD1) en hipertensión arterial esencial: Posible rol etiopatogénico