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Several interventions exist for overcoming high platelet reactivity (HPR) on clopidogrel therapy. The goal of this study was to identify strategies that improve inhibition of platelet reactivity in clopidogrel nonresponders with or without loss of function CYP2C19 genotypes, resulting in platelet reactivity similar to that in responders.

Comparison of antiplatelet treatment in patients with clopidogrel nonresponders with or without carriage of <i>CYP2C19</i> polymorphism