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The effects on placentophagia of strain, reproductive condition, and strain of placenta donor were observed in nonpregnant mice. Mice of the C57BL/6By and BALB/cBy strains were exposed to placentas of either strain after either no previous parturitional experience, one parturitional experience without nursing experience, or one parturitional experience with nursing experience. There was a significant effect of strain, a significant effect of reproductive condition, but no significant effect of strain of placenta donor. There was a significant interaction between strain and reproductive condition, but no significant interactions with placenta strain. It was inferred that the ability of a mouse to acquire and utilize relevant stimuli during and after parturition, in order to produce an emancipation of placentophagia from the physiological controls associated with parturition, is influenced by genotype. Placentophagia (the eating of placenta, normally at parturition) is a little understood phenomenon, despite the fact that it occurs in most mammalian species. General hunger or food deprivation immediately prior to parturition has been eliminated as a motivating factor in the initiation of placentophagia in the rat (Kristal & Wampler, 1973). Furthermore, in the absence of previous parturitional experience, the lateral hypothalamic area of the rat has been shown to exert a major influence on the control of placentophagia, in that damage to that area eliminates placentophagia at parturition, as well as other kinds of ingestive behavior. After parturitional experience has been acquired, damage to the lateral hypothalamic area eliminates homeostatic food and water intake, but placentophagia persists (Kristal, 1973). Likewise, nonpregnant female rats with prior parturitional experience will eat placenta following lateral hypothalamic area damage, even though homeostatic feeding and drinking have been eliminated. The incidence of placentophagia among inexperienced female rats with or without lateral hypothalamic area damage, in the same study, was found to be extremely low (Kristal, 1973). The number of rats exhibiting placentophagia among nonlesioned virgin females presented with placentas was found to be 16%, whereas the number of multiparous, nonpregnant, nonlesioned females exhibiting placentophagia when presented with placentas was 100% (Kristal, 1973). It is thought that parturitional experience emancipates placentophagia from lateral hypothalamic control, and that the behavior

The effects of strain, reproductive condition, and strain of placenta donor on placentophagia in nonpregnant mice