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not yet been precisely defined in humans. The purpose of this study was therefore to analyze findings of electronystagmography (ENG) quantitatively in a patient with a posterior fossa meningioma mainly compressing the posterior vermis. A-70-year-old woman had often experienced a paroxysmal positional dizziness (or a faint sensation) on raising her head from prone position or on turning round, accompanied with no auditory symptoms with recent onset. Such dizziness usually resolved in a moment even though the provoking position was maintained. At our outpatient clinic, no induced nystagmus was observed by positioning or positional changing tests. MRI disclosed a meningioma approximately 2 cm in diameter to the left of the midline at the posterior fossa. The tumor seemed to compress the surface of the cerebellar vermis, mainly the culmen (Lobulus-V) and declive (VI). The other parts of the vestibular-cerebellum (such as flocculus, nodulus or uvula) and the brainstem were apparently not affected. The characteristic findings of the ENG were as follows: (1) lateral beating nystagmus (rightwards>leftwards) in the dark; (2) rebound nystagmus; (3) impairment of vertical smooth pursuit, while the horizontal pursuit was preserved; (4) upbeating nystagmus mainly during shifting eyes rightward or upward in the dark; (5) abnormal eye movements such as double saccadic pulses or ocular flutter were frequently intermingled both in the light and dark; (6) The peak slow phase velocity of OKN was mildly reduced, while frequencies of the slow phase velocity of OKN were preserved; (7) the left OKAN was within normal limits, while the right OKAN (slow phase was left) was not so inhibited and prolonged; and (8) vestibular caloric nystagmus was well induced bilaterally. Both sides of visual suppression of the caloric nystagmus were well preserved. Among these findings, it would be presumed that 原 著

Electronystagmographical studies of a posterior fossa meningioma which mainly compressed the cerebellar vermis