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Infectious diseases are the result of a complex interaction between the infectious pathogen, environmental conditions and host-dependent factors. Several susceptibility differences could be found among individuals of the same population, exposed to the same infectious agent that could be explained by genetic differences. Equine viral arteritis (EVA) is a disease that affects horses causing a systemic infection with variable symptoms, from subclinical infection to respiratory distress and abortions. Between 10 to 70% of the infected stallions can become persistent carriers and spread the virus through the semen. This difference in susceptibility to infection could be explained by differences in genetic constitution. The genetic association studies allow detecting genes involved in the disease by comparing gene variants in infected and uninfected individuals. The genes of the Major Histocompatibility Complex (MHC) are prime candidates for these association studies, given that they encode proteins that play a role in innate and adaptive immune responses. The aim of the study was to identify possible associations between equine MHC genes and resistance / susceptibility to infection with equine arteritis virus. Blood and serum samples from horses previously classified as seronegative or seropositive to EVA were selected. ELA-DRA and TNF-α genes

Determinación de la susceptibilidad/resistencia genética a la infección por el Virus de Arteritis Equina (AVE) mediante el estudio de genes del complejo principal de histocompatibilidad (MHC) equino