English (United Kingdom)

https://curated-unify.zendy.io/wp-json/zendy-region/v1/featured_content/oa?rat=en

https://curated-unify.zendy.io/wp-json/zendy-region/v1/highlighted_journal/

Global: Zendy Plus annual

Presents the access of premium content as premium feature

Premium Content

Presents the keyphrase highlighting as premium feature

Keyphrase Highlighting

Presents the summarisation as premium feature

Summarisation

Insights

Presents the pdf analysis as premium feature

PDF Analysis

Presents the zaia usage as premium feature

ZAIA

Global: Zendy Tools annual

Global: Zendy Free Plan

Global: Zendy Tools monthly

Global: Zendy Plus monthly

480 syndrome and antiphospholipid syndrome in‐ volving the heart and leading to endocardial fi‐ brosis and apical thrombus formation in the left ventricle (LV), as shown on cardiac magnetic resonance (FIGURE 1). Because of a mental disor‐ der, the patient presented poor compliance with anticoagulant (low‐molecular‐weight heparin [LMWH]) and immunosuppressive treatment since the last hospitalization. Transthoracic echocardiography on admis‐ sion showed an extremely large thrombus al‐ most occluding the LV from the mid to apical seg‐ ments, with an impairment of LV contractility. A comparative cardiac magnetic resonance con‐ firmed thrombus enlargement from 22 × 31 mm to 29 × 44 mm and revealed a more advanced Hypereosinophilic syndrome is an uncommon disorder defined as persistent eosinophilia (ab‐ solute eosinophil count >1500/μl) associated with single or multiple organ dysfunction, where other causes of hypereosinophilia have been ex‐ cluded. Cardiac involvement occurs in more than 50% of cases and is a major cause of morbidity and mortality in this patient group.1‐3 The out‐ come depends on the development of endocar‐ dial fibrosis and related complications; there‐ fore, early diagnosis and proper medical treat‐ ment are crucial to prevent disease progression. A 63‐year‐old man was referred to our de‐ partment due to exacerbation of heart failure symptoms and atypical chest pain. Six months earlier he was diagnosed with hypereosinophilic Correspondence to: Marcin Książczyk, MD, Department of Interventional Cardiology and Cardiac Arrhythmias, Medical University of Lodz, ul. Żeromskiego 113, 90‐549 Łódź, Poland, phone: +48 42 639 35 63, email: marcin_ksiazczyk@interia.pl Received: January 10, 2019. Revision accepted: January 30, 2019. Published online: April 25, 2019. Kardiol Pol. 2019; 77 (4): 480‐481 doi:10.33963/KP.14802 Copyright by Polskie Towarzystwo Kardiologiczne, Warszawa 2019 C L I N I C A L V I G N E T T E

kardiologia polska

Intraventricular thrombus progression due to poor compliance with anticoagulant treatment in a patient with hypereosinophilic and antiphospholipid syndromes