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Ash tree disease is caused by an ascomycete fungus Hymenoscyphus fraxineus, which first emerged in 1992, eastern Poland. Site factors, genetic predispositions, and resistance to the pathogen have not been fully described yet. The general aim of the study undertaken was to check the effect of using a new active substance representing benzothiadiazoles, a BTH derivative, namely, N-methyl-N-methoxyamide-7-carboxybenzo(1.2.3)thiadiazole (BTHWA), on ash saplings. A total of 41 ash saplings, aged three to five years, were subjected to this experiment in six variants of treatment. The results of the inoculation with H. fraxineus indicated that the treatment with BTHWA resulted in the limitation of the size of necrotic phloem lesions. Although the lesions were detectable in the cross section, the plants showed no visible signs of infection. The results suggest that H. fraxineus development in ash saplings can be slowed down or even completely stopped through triggering plant resistance by BTHWA.

A Novel Plant Resistance Inducer for the Protection of European Ash (Fraxinus excelsior L.) against Hymenoscyphus fraxineus—Preliminary Studies