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SKIP, a component of the spliceosome, is involved in numerous signaling pathways. However, there is no direct genetic evidence supporting the function of SKIP in defense responses. In this paper, two  SKIPs , namely,  SlSKIP1a  and  SlSKIP1b , were analyzed in tomato. qRT-PCR analysis showed that the  SlSKIP1b  expression was triggered via  Pseudomonas syringae  pv.  tomato  ( Pst ) DC3000 and  Botrytis cinerea  ( B. cinerea ), together with the defense-associated signals. In addition, the functions of  SlSKIP1a  and  SlSKIP1b  in disease resistance were analyzed in tomato through the virus-induced gene silencing (VIGS) technique. VIGS-mediated  SlSKIP1b  silencing led to increased accumulation of reactive oxygen species (ROS), along with the decreased expression of defense-related genes (DRGs) after pathogen infection, suggesting that it reduced  B. cinerea  and  Pst  DC3000 resistance. There was no significant difference in  B. cinerea  and  Pst  DC3000 resistance in TRV- SlSKIP1a -infiltrated plants compared with the TRV- GUS -silencing counterparts. As suggested by the above findings,  SlSKIP1b  plays a vital role in disease resistance against pathogens possibly by regulating the accumulation of ROS as well as the expression of DRGs.

SKIP Silencing Decreased Disease Resistance Against Botrytis cinerea and Pseudomonas syringae pv. tomato DC3000 in Tomato