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Lesion mimic mutants provide ideal genetic materials for elucidating the molecular mechanism of cell death and disease resistance. Here, we isolated a  Glycine max lesion mimic mutant 2-1  ( Gmlmm2-1 ), which displayed a light-dependent cell death phenotype. Map-based cloning revealed that  GmLMM2  encods a coproporphyrinogen III oxidase and participates in tetrapyrrole biosynthesis. Knockout of  GmLMM2  led to necrotic spots on developing leaves of CRISPR/Cas9 induced mutants. The  GmLMM2  defect decreased the chlorophyll content by disrupting tetrapyrrole biosynthesis and enhanced resistance to  Phytophthora sojae . These results suggested that  GmLMM2  gene played an important role in the biosynthesis of tetrapyrrole and light-dependent defense in soybeans.

Genetic Mapping of a Light-Dependent Lesion Mimic Mutant Reveals the Function of Coproporphyrinogen III Oxidase Homolog in Soybean