English (United Kingdom)

https://curated-unify.zendy.io/wp-json/zendy-region/v1/featured_content/oa?rat=en

https://curated-unify.zendy.io/wp-json/zendy-region/v1/highlighted_journal/

Global: Zendy Tools annual

Presents the keyphrase highlighting as premium feature

Keyphrase Highlighting

Presents the summarisation as premium feature

Summarisation

Insights

Presents the pdf analysis as premium feature

PDF Analysis

Presents the zaia usage as premium feature

ZAIA

Global: Zendy Tools monthly

Global: Zendy Plus monthly

Presents the access of premium content as premium feature

Premium Content

Global: Zendy Plus annual

Global: Zendy Free Plan

Stress-induced proline accumulation in plants is thought to result primarily from enhanced proline biosynthesis and decreased proline degradation. To identify regulatory components involved in proline transport, we screened for  Arabidopsis thaliana  T-DNA mutants with enhanced tolerance to toxic levels of exogenous proline (45 mM). We isolated the   p roline  re sistant 1-1  ( pre1-1 ) mutant and map-based cloning identified  PRE1  as  AMINO ACID PERMEASE1  ( AAP1 , At1g58360), which encodes a plasma membrane-localized amino acid permease.  AAP1  expression is induced by salt stress and abscisic acid, but not by proline. In  pre1-1  mutants, a 19-nucleotide deletion in the  AAP1  coding region produced a premature stop codon. When grown on proline-containing medium,  pre1-1  mutants accumulated significantly less proline than did the wild type. Under salt stress, proline uptake decreased significantly in  pre1-1  mutants. By contrast, proline uptake increased significantly in the wild type. These results suggest that AAP1 functions in the increase of proline uptake during salt stress. In addition, proline uptake promotes salt tolerance in Arabidopsis seedlings. We conclude that plants can increase proline accumulation by AtAAP1-mediated proline uptake from exogenous source, which help to improve the salt tolerance of seedlings.

Arabidopsis AMINO ACID PERMEASE1 Contributes to Salt Stress-Induced Proline Uptake from Exogenous Sources