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Over the past years, small species of Tau protein have been increasingly investigated in the field of Alzheimer’s Disease (AD) considering their propensity to induce a synaptic toxicity in neurons before larger aggregates appear. Although scientists got more insight into their effects, their studies usually implied non-physiological conditions with recombinant proteins purified from bacteria ensuring a good concentration but missing post-translational modifications yet well-known to play a key role in AD.

Toxicity assessment of the hyperphosphorylated Tau protein on the neuronal network connectivity