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BACKGROUND OF THE PROJECT: Diets rich in fats have been shown to be detrimental to cognitive performance. Consumption of High fat (HF) diet and associated obesity has influence on cognition. It is assumed that HF diet during pregnancy and lactation is detrimental to the future health of offspring, irrespective of the maternal diet prior to conception. A very limited number of studies indicated that maternal HF diets might be an important trigger that alter brain development and increase the cognitive dysfunctions in the offspring. However, the epidemic of maternal obesity is still relatively new and we don’t have the longitudinal data to know how maternal HF diets affect late-life health of offspring. Investigations into the precise mechanisms behind these changes in offspring are now required. With the current prevalence of maternal obesity throughout world, it is vital to examine the impact of the same on the behaviour and physiology of future generations. Recent data showed that it is not simply HF diet that is damaging the brain, but the nutritional status also influence. Recently, low calcium intake has been identified as a potential contributing factor to obesity. New evidence and review of earlier studies supports the view that calcium also plays a role in adipocyte lipid kinetics at the cellular level and in moderating fatness at the population. However, very few studies have investigated the protective role of calcium supplement against HF diet induced neurological disorders especially in early life.

Postnatal neurochemical and behavioral perturbations in rats following prenatal consumption of high fat diet: protective effect of calcium