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Protein kinase C (PKC)- and RhoA/Rho-associated kinase (ROCK) play important roles in arterial sustained contraction. Although depolarization-elicited RhoA/ROCK activation is accepted, the role of PKC in depolarized vascular smooth muscle cells (VSMCs) is a subject of controversy. Our aim was to study the role of PKC in arterial contraction and its interaction with RhoA/ROCK.

Pkcα-Mediated Downregulation of Rhoa Activity in Depolarized Vascular Smooth Muscle: Synergistic Vasorelaxant Effect of Pkcα and ROCK Inhibition